Neurobiology of Disease Common Alzheimer’s Disease Risk Variant within the CLU Gene Affects White Matter Microstructure in Young Adults

نویسندگان

  • Meredith N. Braskie
  • Neda Jahanshad
  • Jason L. Stein
  • Marina Barysheva
  • Katie L. McMahon
  • Greig I. de Zubicaray
  • Nicholas G. Martin
  • Margaret J. Wright
  • John M. Ringman
  • Arthur W. Toga
  • Paul M. Thompson
چکیده

Meredith N. Braskie,1* Neda Jahanshad,1,2* Jason L. Stein,1* Marina Barysheva,1 Katie L. McMahon,4 Greig I. de Zubicaray,5 Nicholas G. Martin,6 Margaret J. Wright,6 John M. Ringman,3 Arthur W. Toga,1 and Paul M. Thompson1 1Laboratory of Neuro Imaging, Department of Neurology, 2Medical Imaging Informatics, Department of Radiology, and 3Mary S. Easton Center for Alzheimer’s Disease Research, Department of Neurology, University of California, Los Angeles, School of Medicine, Los Angeles, California 90095, 4Centre of Advanced Imaging and 5School of Psychology, University of Queensland, Brisbane, Queensland 4072, Australia, and 6Queensland Institute of Medical Research, Brisbane, Queensland 4029, Australia

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Common Alzheimer's disease risk variant within the CLU gene affects white matter microstructure in young adults.

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A gene-brain-cognition pathway for the effect of an Alzheimer׳s risk gene on working memory in young adults.

Identifying pathways by which genetic Alzheimer׳s disease (AD) risk factors exert neurocognitive effects in young adults are essential for the effort to develop early interventions to forestall or prevent AD onset. Here, in a brain-imaging cohort of 59 young adults, we investigated effects of a variant within the clusterin (CLU) gene on working memory function and gray matter volume in cortical...

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Genetically-mediated Grey and White Matter Alteration in Normal 
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تاریخ انتشار 2011